By Prof Dr. Herwig Hahn von Dorsche, Dr. Harald Schäfer, Dr. Milan Titlbach (auth.)
The booklet supplies a overview of the state of the art of modern investigations of the adipositas diabetes syndrome of sand rats stored below laboratory stipulations and given numerous vitamin regimes. Measurements of particular actions and flux charges in carbohydrate and fats metabolism decided via various analytical equipment in vitro and in vivo supply insights into the pathophysiology and pathobiochemistry of other types of the diabetic syndrome which can't be acquired in people.
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Additional info for Histophysiology of the Obesity-Diabetes Syndrome in Sand Rats
In a state in which increased adrenal medullary activity is to be expected. , under conditions where the in vitro glucose threshold of insulin secretion is lowered below 5 mM glucose if the results of in vitro measurements can be used for interpretation of in vivo conditions. Aside from problems of interpretation, it is remark that alterations in adrenergic interactions after exclusion of the adrenal medullary glands can prevent diet-mediated hyperinsulinemia in sand rats under stress conditions, possibly also by way of central nervous regulation.
This has recently been indirectly confirmed by Sud a and Hashimoto (1978) using Grimelius' (1968) silver impregnation method. These islet cells situated outside the actual islets must be considered responsible for the presence of islet hormones in the pancreatic juice (Prinz et al. 1980). The a-granules of the glucagon cells have recently come under scrutiny in connection with the hormone content of the islet cells. 1 x 105 molecules of glucagon. 0 X 105 molecules). The secretion of hormones from the islet cells depends on the motion of the granules.
It was also found that the difference in islet size between the NH and the diabetic groups was significant at the 99% level. We can thus state that increasing manifestation of diabetes mellitus in the sand rat is associated with enlargement of the islets (Fig. 13). This is due to hyperplasia rather than to hypertrophy. As differential A and B cell staining revealed, hyperplasia is restricted almost exclusively to the B cells. 37 Fig. 14. Well-granulated A cell from the NH group with lipofuscin granules (dark), x12500 Fig.