Apoptosis by S. Tanuma, D. Shiokawa (auth.), Prof. Dr. Y. Kuchino, Prof.

By S. Tanuma, D. Shiokawa (auth.), Prof. Dr. Y. Kuchino, Prof. Dr. W. E. G. Müller (eds.)

Apoptosis performs a valuable position within the rules of phone proliferation. Disruption of this keep an eye on mechanism could cause severe human ailments corresponding to encephalomyelitis and melanoma. hence, knowing of the molecular mechanisms of apoptotic mobile dying should still bring about basic advances within the treatment of those diseases.

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1992). Thus, FeLV immunosuppression could be mediated through activation of endonucleases in thymocytes unprotected by activated protein kinase C (Dezzutti et al. 1989; Rojko et al. 1992). , already fully differentiated) cells like resting lymphocytes are somewhat protected from apoptosis. However, cortical thymocytes and antigen-stimulated lymphocytes proliferate and differentiate to acquire function. Cells undergoing activation or mitosis may be especially susceptible to inappropriately timed apoptosis induction by FeLV; certainly T-cell killing by HIV and SIV is facilitated by prior activation (Ameisen and Capron 1991; Laurent-Crawford et al.

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